Portal Hypertension Puns

How schistosomiasis can cause portal hypertension?

SAAG (serum-ascites albumin gradient) ≥1.1 g/dL (indicates portal hypertension) Cardiac ascites, cirrhosis, Budd-Chiari syndrome <1.1 g/dL (absence of portal hypertension) TB, peritoneal carcinomatosis, pancreatic ascites, nephrotic syndrome

Why is a decrease in oncotic pressure capable of contributing to ascites but cannot cause ascites on its own? dr. Ryan says that portal HTN can cause ascites on its own but decrease in oncotic pressure can contribute to ascites but cannot CAUSE ascites How come ?

Anyone knows?

EDIT: I also wonder if in an early cirrhotic patients (or perhaps those with advanbced fibrosis), weight loss, exercise and diet could reverse portal hypertension?

I read literature that showed limiting PRBC transfusions lead to 45% increase in mortality rate with GI bleeds and portal hypertension. One study cited briefly mentioned not transfusing if hemoglobin is 8 or above in patients with very specific comorbidities. I understand how volume can increase pressure and cause a rebleed. But I don’t understand why hemoglobin was a parameter to transfuse. Can hemoglobin increase portal hypertension?

Age - 59

Height - 5 foot 6 inches / 166

Weight - 82KG

Gender - Female

Non-smoker/No Drinking

Medical Condition- Diabetes, NAFLD(Non-Alcoholic Fatty liver Dieses), Cirrhosis, Hypertension

Hello Docs,

I am writing on behalf of my mother. She diagnosed with cirrhosis around 2 and a half year ago and then doctor seal the cirrhosis and it was running fine for 2 years. But Last month she again puked some blood and then we did endoscopy and diagnosis was Esophageal Varices so the doctor put some ring in the esophagus. Now after a month, same thing happened. She got blood in puke as well as a stool. We rushed to hospital and doctor did a colonoscopy /endoscopy and they found nothing. But later with a CT scan, they found its gastric varices. So now we consulted 2 different intervenous radiologists and they both have a different opinion. 1 doctor suggesting that we should do BRTO (Balloon-occluded retrograde transvenous obliteration) and the other is saying we should go with TIPS(Transjugular intrahepatic portosystemic shunt). Both have merits and demerits. I tried to do research on google but I could not find good sources. It would be great help if any of you can give some insights on what to choose. Thanks in advance and happy holidays.

CT scan Report

Reports

https://imgur.com/0mQrqcE

Endoscopy Report

https://imgur.com/IGvKhil

My Uncle has been a heavy drinker everyday for over 30 years, and the harsh reality of its damage to his liver and brain are catching up with him.

He's undergoing a lot of tests right now to determine how extensive the damage is, but I know it's not good. The portal hypertension is a result of severe cirrhosis of his liver. He's jaundiced, has been vomiting/diarrhea nonstop for a week, and we've heard from his wife that she's noticed signs of mental confusion and dementia-like symptoms over the past couple months.

Uncle Rick is only 63 years old, and if he survives this trip to the hospital I can only hope/pray that he stops drinking to save his own life.

He's the last of my Dad's side of the family, and he's been pretty distant the past few years. I feel so bad that he might lose his brother to this disease...

It's a very real reminder that this substance is deadly if you abuse it. This also makes me feel grateful that I'm on the right track with my own recovery--for my own sake, and so I can be emotionally supportive for my family.

I wanted to share to get some of this out of my own head, and to welcome any feedback/suggestions from those of you that have experience with loved ones who've had hypertension, cirrhosis, or WKS (aka wet brain disease).

Sorry for the dark/harsh reality with this post, but it's not an exaggeration when people say that this is a life or death situation.

IWNDWYT

First Post 26 NOV 2013

Second Post 3 DEC 2013

Third Post 4 Dec 2013

Fourth Post 10 DEC 2013

Fifth Post 17 DEC 2013

Sixth Post 19 DEC 2013

Seventh Post 20 DEC 2013

Eighth Post 23 DEC 2013

Ninth Post 30 Dec 2013

Tenth Post 31 Dec 2013

Eleventh Post 14 Jan 2014

SubPost Requesting help and Summary

Today's, 22 Jan 2014, earlier update

So the standing diagnosis is a combination of microvesicular steatosis, portal hypertension and hepatic edema. I forgot to mention that she also is still fighting a URI.

I went in to take her to Dr Dorn after I made my last post. He drew off about 22 ml of fluid. Today she was 775 grams. Only a few weeks ago she was 650 grams. She is retaining more and more fluid and has swelling everyplace, not just her peritoneal cavity. Her vent, for instance, is extremely puffy. There is absolutely nothing we can do about the tissue edema. There is no such thing as a diuretic for lizards. All we can do is draw off fluid from her belly cavity.

We've worked out a plan of treatment. No more bugs. At all. Low to no fat in her diet and low protein as well. I'm going to look into powdered iguana food or another similar, insectivorous reptile diet. The Oxbow Critical C

I'll give you the only information my mother will give me. I'm 21 and no one tells me what's going on until someone's in the hospital, and even then, they give me the bare minimum. I need your help figuring out what's wrong with my dad.

My dad was in the hospital in Oct 2012 because he was bleeding from "lesions" in his esophagus and stomach. My mom just text me this morning to tell me that he's back in the hospital because he's bleeding from said lesions again. She said:

"That's what he had before. The lesions in his esophagus and stomach...that's what made him bleed... we don't know why it started again... they are caused from portal hypertension in his organs like stomach and liver ... high blood pressure in the organs... we had never heard about that until last time he was here"

Can anyone explain this situation to me? Tell me the severity of it, because she sure as hell won't.

Edit: I'll be getting more information after the doctors run tests this afternoon and will update then. I'd like to keep this post "unanswered" until then in case the info I get needs to be explained. Thanks to anyone who can offer any explanations. This means a lot to me.

Edit 2: I went up to the hospital at 6:30 and stayed until 10:30 last night. My dad was knocked out and pretty dopey when he woke up, but as far as I can tell they're just going to be changing up his meds and trying basically the same treatment as last time. Thank you to everyone who answered my question and helped me out!

I want to thank this community and particularly those members that reached out and spoke to me personally about my situation and about theirs. Cirrhosis isn’t easy to deal with, and just knowing you (or a loved one) has been diagnosed can have serious repercussions on your current life, and future outlook. I apologize for not responding to anyone for a while, but this should help clear up some of the reasons why I have been unavailable. Thank you again for your kind words of support, and I wish you all happiness and health for yourselves and your families.

For those just tuning in: I was diagnosed with decompensated cirrhosis in December 2020. I noticed some slight jaundice in the eyes, and edema in the legs. There were other minor indicators that I realized on later (bloody nose, bleeding gums, hair loss) but my diagnosis was only given after blood tests revealed the extent of my condition. Generally speaking, at that time in December, I was just generally feeling unwell, but had no severe external signs of how bad my health had become.

As time went on, I found myself going to the hospital more and more. Every time, it was because my blood sodium levels were dropping to low. This is a lethal situation. There are two components to sodium levels that are measured. Tissue sodium, and blood/fluid/serum levels. Sodium in the tissues accumulates and leads to edema as the cells retain water (compounded by the pressure changes caused by liver damage [portal vein hypertension] and leaking fluids). Sodium in the blood is much more complicated and is known for being very difficult to control. I had both problems. With low sodium levels, you can’t just eat more sodium and with edema, the typical approach to controlling it is with diuretics and a low sodium diet, along with fluid restriction. These two problems (edema and low blood sodium levels) are at odds with each other. The diuretics will help eliminate the excess fluid in your limbs, but it will also deplete the sodium in your blood.

Why is sodium so important? Among the many things it does, it also regulates your nervous system and the chemical/electrical systems that it is made of. If your sodium gets low enough, you will begin to hallucinate, go into a coma, and eventually die of a heart attack because your heart’s electrical signals are no longer functioning properly.

A few weeks ago I was in the hospital for the 5th time. My sodium was again, extremely low (112 if you’re interested). The doctors and nur

Hey all! My surgery preceptor asked me to look up "celestial (maybe selental?) hypertension" and tell him about it tomorrow.... Problem is when I google that nothing comes up. I must have the first word wrong

We were on the topic of portal hypertension if that helps!

Thank you!!

Just occurred to me to look for this sub, and I'm glad to find it. I am really uncomfortable discussing my diagnosis because I feel like all people hear is "I drank myself to death." My drinking didn't help, but my diagnostic formulation isn't that clear cut. There were concurrent problems with prescriptions and comorbid conditions. I was diagnosed January 2 of this year. I presented to an urgent care clinic and was sent to the ER by ambulance. My presenting problem was anemia--I just didn't know how bad. Turns out it was post-hemorrhagic anemia (I was given bad advice from a doctor that failed to adequately address ongoing bleeding issues). I had a hemoglobin count of 2.3 (24 hour survival rate of 50%). I have had a lot of blood transfusions. At that time it was found that I had dysfunction in my liver, kidneys, spleen, pancreas, and gall bladder. The relative onset of these issues has not been determined. In February, never having had blood sugar issues before, I started showing up at the hospital with glucose levels of 400 to 500+, so I have diabetes to deal with as well.

In positive news, in January I was told I had a MELD score of 22, and decompensated cirrhosis. As of March my MELD score is 7, my cirrhosis is compensated, and my hep. doc says my liver function is "almost normal." No asceties, little to no evidence of varices, mild portal hypertension.

I am curious about what kind of prognosis information y'all have gotten from your docs. When I asked my hep doc in January what my prognosis is (survival rates, anticipated disease course, etc.) And he said that the goal for the first 6 to 12 months was just to keep me alive. We can talk prognosis after that. Creeping up on 6 months, and I think things are much better. Has anyone else had experience with either waiting for a prognosis (or getting one)?

Sorry to show up out of the blue and just dump my purse on the table, but it is such a relief to find a setting where it seems reasonable to pour all of this out.

Thank you for existing.

Clark County COVID-19 positives as of Thursday, June 17th:

255,153, ⬆️241 from 254,912 (6/16)

• Clark County Positivity Rate: 3.6% (6/16), 3.5% (6/15), 3.5% (6/14)

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14-day tracking estimates:

• Active Cases: 3016
• Hospitalized: 222

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SNHD reports 95.8% (244,499, ⬆️170 from 244,329 (6/16)) of cases have recovered.

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SNHD weekly flu snapshot (week 19: 5/9 thru 5/15):

Age / Deaths / Hospitalized

• 0-4 / 0 / 1
• 5-17 / 0 / 0
• 18-24 / 0 / 3
• 25-49 / 0 / 7
• 50-64 / 0 / 12 ⬆️1
• 65+ / 6 / 27
• Total / 6 / 50 ⬆️1
• 2019-2020 Cases: 1487
• 2018-2019 Cases: 972

50 influenza-associated hospitalizations and 6 deaths. Approximately 32% area ER and urgent care visits were adults (age 18-44) for flu symptoms. Influenza A is the dominant strain.

-------

Total Hospitalized: 13,133, ⬆️22 from 13,111 (6/16)

• ICU: 4400, ⬆️3 from 4397 (6/16)
• Intubated: 2146, ⬆️1 from 2145 (6/16)
• Deceased while hospitalized: 3778, ⬆️7 from 3771 (6/16)

*Hospitalized excludes deaths

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Total Deaths: 4459, ⬆️7 from 4452 (6/16)

(3114 with underlying medical conditions)

Not mutually exclusive conditions:

• Hypertension 2034⬆️
• Immunocompromised 151
• Chronic Heart Disease 950⬆️
• Chronic Liver Disease 109
• Chronic Kidney Disease 679⬆️
• Diabetes 1479⬆️
• Neurologic/Neurodevelopmental 360
• Chronic Lung Disease 764⬆️
• Historically Healthy 270
• Other 1235⬆️

-------

Positive Results Age Range Breakdown:

• 0-4 5268⬆️8 2.06%
• 5-17 24527⬆️24 9.61%
• 18-24 31946⬆️30 12.50%
• 25-49 116710⬆️126 45.80%
• 50-64 49318⬆️37 19.30%
• 65+ 27116⬆️16 10.60%
• Unknown 89 0.03%

MIS-C Cases 70

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School Cases (6/17)

Total: 1670 ⬆️20; Past 2wks: 34 ⬆️12

• Staff 762 ⬆️

I'll just be honest. I'm in my last semester and taking 22 credits. Many days I'm completely losing my shit due to the stress. When it gets too bad and I feel like punching several holes in my wall, I have a couple beers. It helps a lot. I don't know if this means I'm instilling alcoholic tendencies into my personality, but damn. It keeps me sane in this incredibly intense time, and doesn't seem to be screwing me over either.

Phil

N-Acetylcysteine (NAC)

People have often asked me which among the supplements I take are most important. Invariably my answer includes NAC. To this day I take three 500 mg capsules each and every day and plan on doing so for the rest of my life. You will literally have to pry it from my cold dead hands.

For this reason, I was extremely upset to learn that NAC was being banned in the United States and was no longer available. To my mind, NAC is essential for those with liver disease. It helps boost levels of glutathione which is integral to the liver for the purpose of detoxification.It also has many hepatoprotective benefits and significantly reduces oxidative stress.

I offer this quote from an abstract on the National Institute of Health entitled “Oxidative Stress and Inflammation in Hepatic Diseases: Therapeutic Possibilities of N**-Acetylcysteine”** in an effort to provide rationale for my strong feelings about NAC supplementation.

"The liver has multiple functions and is the principal detoxifying organ, acting in the clearance of pathogens, toxic chemicals and metabolic waste products from the body, also contributing, for the adequate function of other organs. It impacts heavily almost all physiologic systems to maintain homeostasis [1,2,3]. The continuous exposure of the liver to some factors, such as viruses, alcohol, fat, biotransformed metabolites, among others, can cause hepatic injury, which can lead to inflammation and liver degeneration. When the injury is sustained for long time, it can cause chronic liver diseases (CLDs), which occur in multistage processes of fibrosis, cirrhosis and hepatocellular carcinoma (HCC) [1]. Liver disease has a high prevalence in the world. For some patients, complications may occur, including portopulmonary hypertension, hepatorenal and hepatopulmonary syndromes [4].

Liver fibrosis is a wound healing process, which is reversible and results from chronic liver injuries, including those caused by alcohol consumption, chronic viral hepatitis, autoimmune diseases, parasites, metabolic diseases, lipopolysaccharide (LPS) [[5](https:

Hi, I'm sorry to be here and tell you our story. My 63 yo husband was recently diagnosed with decompensated cirrhosis, portal hypertension and HCC. His cirrhosis is combined with the large (6cm)tumour make a transplant his only option. His specialist (who we've only had one phone appointment with) isn't sure if my husband will be eligible for the transplant because of his cancer. But we are still waiting on the biopsy results. My head is spinning. This has all happened so fast and I don't know what to expect. Hubby spent 3 weeks in the hospital and has only been home 5 days. He feels crappy today and I'm scared he's worsening.

Sudden Lee

Go post NSFW jokes somewhere else. If I can't tell my kids this joke, then it is not a DAD JOKE.

If you feel it's appropriate to share NSFW jokes with your kids, that's on you. But a real, true dad joke should work for anyone's kid.

Mods... If you exist... Please, stop this madness. Rule #6 should simply not allow NSFW or (wtf) NSFL tags. Also, remember that MINORS browse this subreddit too? Why put that in rule #6, then allow NSFW???

Please consider changing rule #6. I love this sub, but the recent influx of NSFW tagged posts that get all the upvotes, just seem wrong when there are good solid DAD jokes being overlooked because of them.

Thank you,

A Dad.

So far nobody has given me a straight answer

Because a toothbrush works better

Had to ground him until he could conduct himself properly.

I am currently in the hospital. I had a back operation yesterday. The surgical nurse came in my room and started asking questions about my back. She asked me if I had any falls during the last year. I responded just one. It was after summer.

She laughed and said in 20 years of doing this she never was told that joke.

Indian places are naan profit, Vietnamese places are pho profit.

..... Will get a reward.

First Post 26 NOV 2013

Second Post 3 DEC 2013

Third Post 4 Dec 2013

Fourth Post 10 DEC 2013

Fifth Post 17 DEC 2013

Sixth Post 19 DEC 2013

Seventh Post 20 DEC 2013

Eighth Post 23 DEC 2013

Ninth Post 30 Dec 2013

Tenth Post 31 Dec 2013

Eleventh Post 14 Jan 2014

SubPost Requesting help and Summary

I heard from Dr Wagner, who is an exotics specialist beyond normal exotics specialists. He is at the University of Pittsburgh and also has worked for/ the Pittsburgh Zoo as well as The National Aviary. If anybody is going to figure her out, it's him. He did the ultrasound a week ago tuesday and took a liver biopsy guided by it.

The first and only good news is that it is not cancer.

The bad news is that it is still very, very bad.

He has diagnosed her with microvesicular steatosis of the liver, portal hypertension and hepatic edema. He prognosis is not excellent.

-1. Microvesicular Steatosis of the liver

• Steatosis basically means "fat change" and deals with the abnormal holding of lipids (fats) in a cell. The body is unable to rid cells of these lipids and becomes more impaired over time. Excessive lipids build up in vesicles and actually displace the fluid (cytoplasm) inside the cells themselves.

• Microvesicular specifically means that the vesicles aren't large enough to mov

Clark County COVID-19 positives as of Monday, June 14th:

254,376, ⬆️98 from 254,278 (6/13) [⬆️68 6/13, ⬆️241 6/12, ⬆️215 6/11]

• Clark County Positivity Rate: 3.4% (6/13), 3.4% (6/12), 3.4% (6/11)

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14-day tracking estimates:

• Active Cases: 2971
• Hospitalized: 216

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SNHD reports 95.9% (243,932, ⬆️73 from 243,859 (6/13)[⬆️66 6/13, ⬆️199 6/12, ⬆️194 6/11]) of cases have recovered.

-------

SNHD weekly flu snapshot (week 19: 5/9 thru 5/15):

Age / Deaths / Hospitalized

• 0-4 / 0 / 1
• 5-17 / 0 / 0
• 18-24 / 0 / 3
• 25-49 / 0 / 7
• 50-64 / 0 / 12 ⬆️1
• 65+ / 6 / 27
• Total / 6 / 50 ⬆️1
• 2019-2020 Cases: 1487
• 2018-2019 Cases: 972

50 influenza-associated hospitalizations and 6 deaths. Approximately 32% area ER and urgent care visits were adults (age 18-44) for flu symptoms. Influenza A is the dominant strain.

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Total Hospitalized: 13,045, ⬆️4 from 13,041 (6/13) [⬆️4 6/13, ⬆️32 6/12, ⬆️21 6/11]

• ICU: 4386, NC from 4386 (6/13) [⬆️1 6/13, ⬆️6 6/12, ⬆️7 6/11]
• Intubated: 2144, NC from 2144 (6/13) [⬆️1 6/13, ⬆️2 6/12, ⬆️3 6/11]
• Deceased while hospitalized: 3762, NC from 3752 (6/13) [NC 6/13, ⬆️3 6/12, ⬆️3 6/11]

*Hospitalized excludes deaths

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Total Deaths: 4443, NC from 4443 (6/13) [⬆️0 6/13, ⬆️3 6/12, ⬆️1 6/11]

(3091 with underlying medical conditions)

Not mutually exclusive conditions:

• Hypertension 2013
• Immunocompromised 151
• Chronic Heart Disease 946
• Chronic Liver Disease 109
• Chronic Kidney Disease 672
• Diabetes 1467
• Neurologic/Neurodevelopmental 359
• Chronic Lung Disease 755
• Historically Healthy 270
• Other 1219

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Positive Results Age Range Breakdown:

• 0-4 5250⬆️1 2.06%
• 5-17 24466⬆️17 9.62%
• 18-24 31849⬆️12 12.50%
• 25-49 116434⬆️43 45.80%
• 50-64

Clark County COVID-19 positives as of Monday, May 24th:

250,169, ⬆️86 from 250,083 (5/23) [⬆️74 5/22, ⬆️178 5/21, ⬆️258 5/20]

• Clark County Positivity Rate: 4.5% (5/23), 4.6% (5/22), 4.7% (5/21)
• SNV Hospital Capacity: 73% (5/20), 75% (5/19), 74% (5/18)

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14-day tracking estimates:

• Active Cases: 3358
• Hospitalized: 227

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SNHD reports 95.9% (239,871, ⬆️104 from 239,767 (5/23) [⬆️104 5/22, ⬆️185 5/21, ⬆️275 5/20]) of cases have recovered.

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SNHD weekly flu snapshot (week 19: 5/9 thru 5/15):

Age / Deaths / Hospitalized

• 0-4 / 0 / 1
• 5-17 / 0 / 0
• 18-24 / 0 / 3
• 25-49 / 0 / 7
• 50-64 / 0 / 12 ⬆️1
• 65+ / 6 / 27
• Total / 6 / 50 ⬆️1
• 2019-2020 Cases: 1487
• 2018-2019 Cases: 972

50 influenza-associated hospitalizations and 6 deaths. Approximately 32% area ER and urgent care visits were adults (age 18-44) for flu symptoms. Influenza A is the dominant strain.

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Total Hospitalized: 12,680, ⬆️1 from 12,579 (5/23) [⬆️4 5/22, ⬆️13 5/21, ⬆️35 5/20]

• ICU: 4301, ⬆️1 from 4300 (5/23) [⬆️1 5/22, ⬆️3 5/21, ⬆️4 5/20]
• Intubated: 2105, NC from 2105 (5/23) [⬆️1 5/22, ⬆️1 5/21, ⬆️2 5/20]
• Deceased while hospitalized: 3705, NC from 3705 (5/23) [NC 5/22, ⬆️9 5/21, ⬆️2 5/20]

*Hospitalized excludes deaths

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Total Deaths: 4383, NC from 4383 (5/23) [NC 5/22, ⬆️7 5/21, ⬆️1 5/20]

(3002 with underlying medical conditions)

Not mutually exclusive conditions:

• Hypertension 1937
• Immunocompromised 148
• Chronic Heart Disease 900
• Chronic Liver Disease 105
• Chronic Kidney Disease 653
• Diabetes 1428
• Neurologic/Neurodevelopmental 348
• Chronic Lung Disease 731
• Historically Healthy 269
• Other 1163

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Positive Results Age Range Breakdown:

• 0-4 5169 2.07%
• 5-17 24042⬆️16 9.61%
• 18-24

Clark County COVID-19 positives as of Wednesday, June 9th:

253,506, ⬆️209 from 253,297 (6/8)

• Clark County Positivity Rate: 3.4% (6/8), 3.5% (6/7), 3.4% (6/6)

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14-day tracking estimates:

Active Cases: 2933

Hospitalized: 233

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SNHD reports 95.9% (243,183, ⬆️149 from 243,034 (6/8)) of cases have recovered.

-------

SNHD weekly flu snapshot (week 19: 5/9 thru 5/15):

Age / Deaths / Hospitalized

• 0-4 / 0 / 1
• 5-17 / 0 / 0
• 18-24 / 0 / 3
• 25-49 / 0 / 7
• 50-64 / 0 / 12 ⬆️1
• 65+ / 6 / 27
• Total / 6 / 50 ⬆️1
• 2019-2020 Cases: 1487
• 2018-2019 Cases: 972

50 influenza-associated hospitalizations and 6 deaths. Approximately 32% area ER and urgent care visits were adults (age 18-44) for flu symptoms. Influenza A is the dominant strain.

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Total Hospitalized: 12,955, ⬆️26 from 12,929 (6/8)

• ICU: 4367, ⬆️12 from 4355 (6/8)
• Intubated: 2137, ⬆️5 from 2132 (6/8)
• Deceased while hospitalized: 3753, ⬆️6 from 3747 (6/8)

*Hospitalized excludes deaths

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Total Deaths: 4435, ⬆️7 from 4428 (6/8)

(3070 with underlying medical conditions)

Not mutually exclusive conditions:

• Hypertension 1989⬆️
• Immunocompromised 151
• Chronic Heart Disease 936⬆️
• Chronic Liver Disease 108⬆️
• Chronic Kidney Disease 667⬆️
• Diabetes 1457⬆️
• Neurologic/Neurodevelopmental 354⬆️
• Chronic Lung Disease 749⬆️
• Historically Healthy 269
• Other 1207⬆️

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Positive Results Age Range Breakdown:

• 0-4 5235⬆️2 2.07%
• 5-17 24382⬆️32 9.62%
• 18-24 31741⬆️22 12.50%
• 25-49 116002⬆️100 45.80%
• 50-64 49072⬆️32 19.40%
• 65+ 26985⬆️21 10.60%
• Unknown 89 0.04%

MIS-C Cases 69

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School Cases (6/9)

Total: 1613 NC; Past 2wks: 3 ⬇️1

• Staff 739 NC; 1 NC
• Stud

Clark County COVID-19 positives as of Thursday, May 20th:

249,573, ⬆️278 from 249,295 (5/19)

• Clark County Positivity Rate: 4.9% (5/19), 4.9% (5/18), 5.0% (5/17)
• SNV Hospital Capacity: 70% (5/16), 72% (5/15), 74% (5/14)

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14-day tracking estimates:

• Active Cases: 4215
• Hospitalized: 260

-------

SNHD reports 95.8% (239,203, ⬆️263 from 238,940 (5/19)) of cases have recovered.

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SNHD weekly flu snapshot (week 18: 5/2 thru 5/8):

Age / Deaths / Hospitalized

• 0-4 / 0 / 1
• 5-17 / 0 / 0
• 18-24 / 0 / 3 ⬆️2
• 25-49 / 0 / 7
• 50-64 / 0 / 11 ⬆️1
• 65+ / 6 / 27 ⬆️1
• Total / 6 / 49 ⬆️2
• 2019-2020 Cases: 1485
• 2018-2019 Cases: 969

49 influenza-associated hospitalizations and 6 deaths. Approximately 40% area ER and urgent care visits were adults (age 18-44) for flu symptoms. Influenza A is the dominant strain.

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Total Hospitalized: 12,627, ⬆️51 from 12,576 (5/19)

• ICU: 4292, ⬆️11 from 4281 (5/19)
• Intubated: 2101, ⬆️4 from 2097 (5/19)
• Deceased while hospitalized: 3694, ⬆️5 from 3689 (5/19)

*Hospitalized excludes deaths

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Total Deaths: 4375, ⬆️5 from 4370 (5/19)

(2994 with underlying medical conditions)

Not mutually exclusive conditions:

• Hypertension 1931⬆️
• Immunocompromised 148
• Chronic Heart Disease 898⬆️
• Chronic Liver Disease 105
• Chronic Kidney Disease 649⬆️
• Diabetes 1424⬆️
• Neurologic/Neurodevelopmental 345⬆️
• Chronic Lung Disease 729⬆️
• Historically Healthy 270
• Other 1153⬆️

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Positive Results Age Range Breakdown:

• 0-4 5165⬆️3 2.07%
• 5-17 23960⬆️24 9.60%
• 18-24 31285⬆️19 12.50%
• 25-49 114101⬆️139 45.70%
• 50-64 48333⬆️57 19.40%
• 65+ 26644⬆️36 10.70%
• Unknown 85 0.03%

MIS-C Cases 66

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**Sc