Since SCP is at a much lower frequency than most EEG measurements, I'm having trouble finding any reasonably priced devices that can measure it accurately.
The best I've found so far is a package from Thought Technologies, at an eye-watering $4,428.
The gains from neurofeedback are permanent, while the gains from tdcs are transient. By training slow cortical potentials you are increasing the overall voltage in your brain on your own and you can quit training once you have reached a satisfactory level.
Has anyone tried this?
Hello, I have made this post because there is something unnoticed going on in this community. This title may sound controversial, so let me explain my concerns.
First, we need to understand the difference between a SubliminaI message and a Supraliminal message. I believe this needs to be addressed because the two get mixed quite alot. So for that, I will tell you the difference below.
• Subliminal •
A SubliminaI is stimuli below conscious threshold. (affirmations & statements that are inaudible to the conscious mind but heard and understood by the subconscious mind)
• Supraliminal •
A Supraliminal is stimuli above conscious threshold but goes unnoticed. (affirmations & statements that are audible to the conscious mind but not understood)
• The misconstruction between the two •
Stimuli that are perceived above the threshold and thus are detected at the level of consciousness are called supraliminal. In contrast, stimuli that are not perceived at the conscious level and thus are not detected are called subliminal: https://images.slideplayer.com/14/4491882/slides/slide_7.jpg
Many creators and individual's assume a subliminaI messaging track works by adjusting the volume, which is inaccurate and wrong. A subliminaI messaging track works by adjusting the frequency to below conscious threshold, allowing the affirmations to go directly to the subconscious mind. The reason why we do this is because we don't want our conscious mind to be aware of the stimuli.
Here is an example below posted by the creator of this subreddit: https://www.reddit.com/r/Subliminal/comments/chaq2k/quick_guide_make_your_own_subliminal/?utm_medium=android_app&utm_source=share - That is not a subliminaI recording due to the stimuli being above conscious threshold.
No disrespect to u/Abernore. They are an amazing lovely individual and I am proud of them for creating a community dedicated to this subject, and also helping so many people improve their life.
• Which is better Supraliminal or Subliminal? •
Supraliminal:
Supraliminal tracks have suggestions that are present to the conscious mind. If you're able to understand what they imply, your conscious mind has the potential to reject that suggestion.
They can create a distraction or disturbance in your daily life.
Subliminal:
Subliminal tracks are more verstile because you do not need to worry about hearing the stimuli. Thanks to this, the chances of the conscious mind rejecting the suggestions are non-existent
... keep reading on reddit ➡I really don't agree with Piracetam being removed from Nootropics Depot as some have suggested. I understand it's not the most economic/ profitable drug due to its bulky size and large dose (as well as a ton of slander) but it's damn effective in those doses, and the father of all nootropics: https://en.wikipedia.org/wiki/Corneliu_E._Giurgea
Things just wouldn't be the same without AMPAergics like Piracetam. What I'm about to present would have a higher entry fee than what some other companies would be capable of, but in the end it would be worth it...
Clausenamide
Clausenamide is basically "herbal piracetam". It's a natural AMPAergic and cholinergic nootropic extracted from the fruit tree Clausena Lansium, currently being studied for cognitive decline in China. There is a language barrier, and the Chinese translation spells "Xanthamide" or "Levoxanthamide" in English, which is likely why not many people know about it.
Seems like it's going through Phase 2 human trials, or may have even passed by this point: https://www.sohu.com/a/116841490_464396
It's a multimillion dollar drug already: "At the end of 2005, the Institute of Drugs transferred Levoxanthinamide to Beijing Qilin Tiansheng Pharmaceutical Co., Ltd. at a price of 24 million yuan." ~3.778 million USD. From what I've read, it was well tolerated.
It shows promise as a nootropic: "The synthetic single enantiomer Clausenamide could improve Ab induced impairment of spatial discrimination, potentiate basic synaptic transmission and HFS-induced LTP on either anesthetized or freely moving rats, and increase cortical ChAT activity, hippocampal synapses and Mossy fiber sprouting. In addition, was 50–100 times more active than the known nootropic drug, Piracetam, and 5–10 times more active than the racemic Clausenamide.":
It being "more active" than Piracetam really means nothing special, but ChAT, the acetylcholine synthesizing enzyme, being enhanced, is unique. In addition to its neurogenic effects, of course.
The anti-dementia drug candidate, (-)-Clausenamide, improves memory impairment through its multi-target effect: https://sci-hub.se/https://www.sciencedirect.com/science/article/abs/pii/S0163725816000036?via%3Dihub
Clausenamide increases thickness of the cerebral cortex and synapses d
... keep reading on reddit ➡Posting this from a burner account for obvious reasons. I have been doing some research regarding PE and wanted to put some of it here to hopefully open a discussion. Apologies for the book I have written below, but it contains some information regarding strategies I have tried (including some that have worked). I do ask that people do not judge, as I am putting deeply personal information here in an attempt for us all to succeed. I'll start with a little about myself
DISCLAIMER: I AM NOT A DOCTOR, AND SOME INFERENCES I MAKE MIGHT BE INCORRECT
I'm a male in his 20's currently struggling with PE. I'm fairly thin, not in great shape but certainly not in bad shape. I drink and smoke (weed) fairly regularly (both are known to cause PE). Since becoming sexually active (mid teens, before drinking / smoking), I have always been fairly quick to finish, however I feel it has likely gotten worse over the past couple years. After being in a non-sexual relationship for the last year, I am now single and getting back to having sex. I quickly noticed that my PE had actually gotten worse, and did not seem to be getting better. This led me to looking deeply in to why this was happening, to see what methods I could use to combat PE. While I am not a doctor, and have no in-depth prior experience with neurology, I work in STEM, and have always found that analytical approaches are the best for solving issues. My below findings have been over the course of about 4 months.
How I got sent down this rabbit hole:
I started with asking "When does this (PE) happen?". Since I don't have a partner to regularly test with, I decided to use porn as a control variable. I would experiment with different types / camera angles, and found that this had some impact on my IELT. Generally, however, I would only last between 1-2 minutes (lowest 50s, highest 1m 45s).
I then decided to cut out porn and any visual / audio stimuli. This increased my IELT by about 1m (so 2m-3m total), however as my end goal is to last longer with a partner, this was not exactly a step in the right direction. I then tried to see if I could distract myself, while not using porn, to see how long I could last. I did this by doing simple math in my head. There were three types of math problems I would do: calculating any single digit number to the nth power (highest possible), doing basic polynomial derivations, and determining whether or not any random 5 digit number was divisible by
... keep reading on reddit ➡The microbes of the gut can connect with the brain to affect anxiety. There are various pathways along which this communication can take place. One is through the major neurotransmitters. The gut microbes such as Bifidobacterium and Bacillus produce the neurotransmitters GABA and dopamine, respectively. The neurotransmitters signal to the nervous system of the gastrointestinal tract, and those signals will be carried to the brain through the vagus nerve. This is demonstrated by the fact that altering the microbiome has shown anxiety and depression reducing effects in mice, but not in subjects without vagus nerves. For example, experiments in which mice were given fructo and galacto oligosaccharide prebiotics and Lactobacillus probiotics have both demonstrated a capability to reduce anxiety. (from wikipedia)
Strong evidence suggests that gut microbiota has an important role in bidirectional interactions between the gut and the nervous system. It interacts with CNS by regulating brain chemistry and influencing neuro-endocrine systems associated with stress response, anxiety and memory function. Many of these effects appear to be strain-specific, suggesting a potential role of certain probiotic strains as novel adjuvant strategy for neurologic disorders. (https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4367209/)
The gut microbiota can be intentionally manipulated to help maintain health and prevent or treat disease. Recent experimental evidence would appear to suggest that alterations to the gut microbiota composition through probiotic treatment could attenuate neuropsychiatric symptoms or even reduce the risk of developing future psychiatric symptoms. For example, treatment with Lactobacillus rhamnosus induced region-dependent changes in GABA expression in the cortical cingulate, This treatment thereby reduced the stress-induced release of cortisol, which in turn reduced anxiety and depression-related behavior. (https://psychscenehub.com/psychinsights/the-simplified-guide-to-the-gut-brain-axis/)
So now in terms of supplements and health foods, what can we do to improve our gut bacteria and subsequently our mental and gastrointestinal health?
Clausenamide
Clausenamide is basically "herbal piracetam". It's a natural AMPAergic and cholinergic nootropic extracted from the fruit tree Clausena Lansium, currently being studied for cognitive decline in China. There is a language barrier, and the Chinese translation spells "Xanthamide" or "Levoxanthamide" in English, which is likely why not many people know about it.
Seems like it's going through Phase 2 human trials, or may have even passed by this point: https://www.sohu.com/a/116841490_464396
It's a multimillion dollar drug already: "At the end of 2005, the Institute of Drugs transferred Levoxanthinamide to Beijing Qilin Tiansheng Pharmaceutical Co., Ltd. at a price of 24 million yuan." ~3.778 million USD. From what I've read, it was well tolerated.
It shows promise as a nootropic: "The synthetic single enantiomer Clausenamide could improve Ab induced impairment of spatial discrimination, potentiate basic synaptic transmission and HFS-induced LTP on either anesthetized or freely moving rats, and increase cortical ChAT activity, hippocampal synapses and Mossy fiber sprouting. In addition, was 50–100 times more active than the known nootropic drug, Piracetam, and 5–10 times more active than the racemic Clausenamide.":
It being "more active" than Piracetam really means nothing special, but ChAT, the acetylcholine synthesizing enzyme, being enhanced, is unique. In addition to its neurogenic effects, of course.
The anti-dementia drug candidate, (-)-Clausenamide, improves memory impairment through its multi-target effect: https://sci-hub.se/https://www.sciencedirect.com/science/article/abs/pii/S0163725816000036?via%3Dihub
Clausenamide increases thickness of the cerebral cortex and synapses density in the hippocampal CA3 region (like a 10% increase in cortical thickness in mice) and of course improves learning and memory: https://sci-hub.se/https://www.tandfonline.com/doi/abs/10.1080/10286029808039843
As far as synthesizing it, it seems that extracting it from herb is a highly expensive process. There was a multi-step extraction employed from another constituent in this paper, which seemed interesting: [https://sci-hub.se/https://pubs.rsc.org/en/content/articlelanding/2009/OB/b901965k](https://sci-hub.se/https://
... keep reading on reddit ➡