I work in a retirement home and one medical histories for a lot of our residents (and one employee) a lot of people kinda just have a-fib with the only other result being some other cardiac/circulation issues plus an irregular heartbeat. However, v-fib is one of two heart rhythms that can result in a heart attack (the other being ventricular tachycardia; v-tac). So why is one life threatening but not the other?
Side note, medical is what I do, so if anyone wants to add other info or correct something I said by all means feel free.
How can I calculate the frequency of atria/ventricle when the rhythm is not regular or when there is fibrillation? on 50mm/s paper.
for example what would the atrial and ventricular frequency be?
https://preview.redd.it/fbms82f44bu61.png?width=2500&format=png&auto=webp&s=72195e9d40725ff43c10ce4fc45c88244615f07c
As a physician, I thought that was an interesting detail.
Ventricular fibrillation, or V-fib, is a heart dysrhythmia that is life-threatening and often leads to cardiac arrest if not defibrillated back into a more adequate and normal rhythm. Thus the importance of having access to an Automatic External Defibrillator or AED in the event you suspect a heart attack.
In this case our AEDs are our ghosts or a friendly guardian!
Cheers
Edit: To all of you Exos saying you donβt have a heart...Cayde had a heart you savages.
Is it possible? I mean the hearts chambers work together donβt they?
Does anyone have no apparent reason for episodes of ventricular fibrillation?
My family has this βconditionβ. Siblings (two males, one female) all have had incidents. Parents do not.
Male 1 - deceased, first and only VF, age 29.
Male 2 - living, First VF age 38, ICD placed. Additional VF episodes aborted by ICD (maybe 3 more incidents since age 38, now 58).
Female (me) βtestedβ age 35 with EP study. Negative results with estimated 5% risk not acceptable, ICD implanted. Age 39, heart started racing, (recorded 285 bpm), ICD fired. Deemed V Tach on its way to VF.
There is no diagnosis other than idiopathic ventricular fibrillation. No way to test, no LQT, no Brugada (I called Dr Brugada (one of the three Brugada brothers who lives in NY) and he looked at one of my brotherβs EKGs and he said it wasnβt Brugada Syndrome. No defect of SCN5A gene. Nothing.
Who can I contact to help us? Our Electrophysiologists keep telling us in a few years weβll know (been telling us this since 1999). Who would be interested in studying this?? Help!!
33 years old male, obese, complaining of shortness of breath, fatigue (sometimes it's extreme), dizziness, palpitations, occasional skipped beats (in the last month I used to have too many of them)
This is an ECG I had about 2 weeks ago and I won't be able to visit a doctor before 2 days from now. So I was just curious if anything is wrong??
I think the first beat recorded in the ECG (the one on the far left) is one of the "skipped beats" or the PVCs (premature ventricular contraction), right??
Do I suffer from Hypertrophic Cardiomyopathy?? or HOCM??
I'm very frightened I will have ventricular fibrillation and die soon
For causing ventricular fibrillation, the required voltage is as follows:
Voltage = 100 mA Γ 300Ξ© = 30 V
Your bodies internal resistance is 300 ohms. Your skin is much higher. If there is a break in the skin, it is much lower. 100 mA or 1/10 of an amp is the threshold for ventricular fibrillation (chaotic heart beat).
What is it and why am I dying from it in Menagerie
Good evening,
I am a newly promoted paramedic. One of the things that my training officer taught me in regards to a fibrillation, is caution in using cardizem. Specifically using cardizem in patients that are in atrial fibrillation for a unknown amount of time and arenβt on a blood thinner. The way it was explained to me is that there can be a clot that forms in the atria while the heart is in afib. When this converts from the cardizem, it can throw that clot and cause bigger issues. This is not a caution listed by the physician that runs my departments paramedics. Itβs not listed in our treatment protocols. Itβs strictly word of mouth hearsay and I just really need clarification. To me itβs pick your poison, if that truly is a risk then you need to weigh the risk. Either throw the clot or the patients heart fails from a unsustainable rhythm. Any insight would be helpful.
