This is literally the dumbest dream I have ever had.
Iβm creating a fun video on how to prepare an UltraTag RBC kit for a school project but need some missing info. Package insert instructions say to use 10-15 units of heparin per ml of blood. (Example: 3ml blood would be 30-45 units of heparin) My question is how many ml of heparin is 30-45 units of heparin? Thanks in advance!
So, tonight I made a heparin drip med error. I know it happened because it was my first time floating to the ED and it was the very begining of the shift and I was distracted and panicked. I am normally a very relaxed person at work. It was hung by the previous nurse who reported to me is was 16 mL/hr. I quickly glanced at the order and saw 16 where the mL/hr is on my floor's order. Okay, it's correct, right. Checked the pump and it was set on 16. Okay, great. We'll, I transfer this pt to the floor and the med nurse calls me an hour later and tells me it's supposed to be 16 units/kg/hr which is 11.5ml/hr.
No harm to pt, PT INR was in therapeutic range. No bleeding, thankfully.
Guys, I'm shook. I am supposed to start as charge in January and now my confidence is in the toilet.
Just had to tell someone.
https://www.bbc.co.uk/news/world-australia-59751702
I found this intriguing as I've seen Heparin pop up a lot lately when researching the microclotting issue of long covid. But this is suggesting a different use for it where it's like a decoy sat up your nose for the virus to attach to instead of entering the body. So could heparin also have the benefit of doing something similar within the blood vessels - like scooping up any viral debris causing the elevated levels of antiplasmin which is stopping the natural breakdown of fibrin?
Anyone know how reluctant or not reluctant a GP might be to prescribe Heparin? I'm currently waiting to get into a long covid clinic (UK) and ask them about it. I doubt a GP would humour me and prescribe it as they wont have a clue what I'm talking about with the microclotting and long covid.
I know that if you can pause, it has to be paused for 2 minutes and you have to waste, but an RN asked us why they could not draw it, and confused my very tiny departmemt overnight. We did just do a venipuncture on the opposite side and follow normal procedures, not wanting the result to be skewed. But after reading, we only found answers about IVs and not ports. We all made the assumption that the port followed the same procedure.
Hi all! I'm having laparoscopic abdominal surgery next month and, due to a DVT I had in 2018 (unprovoked, no genetic factors), my surgeon has let me know that she'll be dosing me with heparin right before surgery, and I'll be on 4 weeks of Lovenox afterwards. The Lovenox makes sense, but everything I've ever seen says to stop thinners before surgery, not start them, so I'm concerned about the heparin prior to surgery. Has anyone here ever had this as part of a care plan/can provide any insight?
Thanks!
Update: was finally able to get ahold of my surgeon and ask, it turns out this particular procedure has a high occurrence of post-surgical clots, so a small prophylactic dose of heparin is recommended prior to surgery regardless of clot history. So TIL, thanks all for your input!
If some has received subq 5,000 units of heparin for prophylaxis then they switch for a heparin drip for PE would you give an IV bolus of heparin?
Anyone with insight/experience how TPA can affect ptt monitoring of Heparin for the first 24-72hrs? I have a patient who was given TPA for PE and immediately started on Heparin drip without checking to see if the baseline ptt was <2x normal. They were given a bolus of Heparin to boot. I'm unsure if their elevated ptt now reflects hemorrhagic transformation and lingering affects of TPA.
Have you guys seen it? Is one flush all it takes or are talking only therapeutic doses of heparin? I get a handful of patients every year and Iβm always surprised when their cvc still works.
I know that APTT is primarily used to assess heparin therapy on a patient and that a heparin contaminated sample will affect the APTT by artificially prolonging it. We use thrombin time in our lab when performing APTT 1:1 mixing studies to detect possible heparin contamination and then treat the specimen with hepzyme. if the thrombin time corrects to normal(ish) range, then we proceed with the mixing study using the treated sample.
But what if other tests were attached to the specimen such as D-dimer, PT-INR, fibrinogen, and some special coags like Lupus screen, protein S activity, Factor VIII activity, etc.???
I've read multiple sources that said that heparin contaminated samples should never be used for any kind of coagulation testing, but some people I work with said it doesn't matter, that it will only affect the APTT and Thrombin Time. But, it's still a contaminated sample, no?? Even though APTT is used to monitor heparin therapy and is sensitive to heparin contamination, I've read that other coagulation testing may be affected, if to a lesser degree.
Any coag experts know if heparin contamination is significant enough if the initial APTT was 70 and Thrombin Time was above the linearity at >60?
I talked to my doc today and some consistent Heparin installations seem to be the best way to go to begin seriously tackling my IC. Only issue is, as a male I'm deathly afraid of catheters for what I imagine would be obvious reasons to most.
He offered to give me a tablet of valium... will this be enough to get me through this procedure in your guys' experience? I don't remember taking valium before and I do not want to be conscious that this will be happening in any sense, feeling it, or anything... it sounds horrible.
Is there anything else I can do or take on my end or even request of the doc to make this process easier and less apparent?
All feedback would be so appreciated!
I take 1 dose of 4500 unit tinzaparin everyday along with a single dose of baby Asprin. Iβve ordered NIPT testing and am just wondering if this medication can really influence the test results? Iβm anxious about them anyway due to previous losses but even more so now.
Hi, I'm a soon-to-be MLT from Germany, my exams are next week :)
I just wanted to ask if anyone of you guys knows why you can't use Lithium-Heparin-Tubes for electrophoresis? It was an exam question a few years back, but it was a multiple choice question and we don't have those anymore, so I'm kinda scared I'm supposed to know the reason.
Thanks in advance! :D
I recently found out that whenever I get pregnant, I'm going to have to give myself Lovenox shots in my stomach every single day until 6 weeks pp because I have a blood disorder. I'm hoping to start TTC in the near future but I'm freaking terrified of these shots. I've read every post I can find in this sub/elsewhere on the internet on other people's experiences with Lovenox, but what I specifically want to know is - how in the world do you get over fear of needles basically overnight? How do you do those first few shots??
I'd love to talk directly to people who have experienced this. I just do not understand how I am suddenly supposed to become okay with sticking a needle in my own stomach. I know I WILL become okay with it eventually because I have to, but how do I start?!! I got a shot at the doctor yesterday and I couldn't even look at it. I was trying to imagine me picking up that needle and putting it in my own stomach, and I just don't know how I'm gonna do it. I'm especially scared because I've read that Lovenox shots can burn. Would love to hear other people's experiences/advice!!
Why is PTT used to measure heparin activity and not PT when antithrombim binds to factor VII too, is it because factor VII is the only factor in the extrinsic pathway and so the intrinsic pathway is affected more due to antithrombin
