Jugular venous pressure Puns

Is this an Emperor's New Clothes situation or are all of you actually seeing something when you're staring at people's necks?

why is there a hugeee y curve descent for constrictive pericarditis but absent y curve in cardiac tamponade?

thanks!!

Man I can't for the life of me get this damn graph down. I know my cardio physio but when trying to combine the motions of the ECG, pressure curve, Ventricular volume curve, and the heart sounds, it doesn't add up to me. Anyone have problems implementing their knowledge into this graph?

Does this sound familiar to anyone?

"Background:We describe a young man who developed exercise-induced intracranial hypertension manifesting as status migrainosus,from chronic posttraumatic jugular vein obstruction after a motorcycle accident. Clinical and radiological findings are described, the potential pathogenesis is discussed and reponse to treatment presented.Methods:This 18 year old non-obese,healthy male patient had a motorcycle accident with left brachial plexus avulsion, asymptomatic left vertebral artery dissection. 4 years after the accident,he had prolonged headache episodes precipitated by strenuous sporting activities. No response to analgesics or triptans was seen.There was no evidence of papillodema and non-contrast head CT was normal.No medication overuse was observed. Lumbar punctures (LP) during headache episodes showed elevation of opening pressures (OP) of > 50cm H20 (normal: 5-20cm) but OP between attacks was normal.He noticed relief of headaches 10-20 minutes following LP. Oral acetazolamide for six months followed by Verapamil for four months did not change the headaches.Cranial CT venography during an episode showed compression of the transverse sinuses. Cervical venous MRA with contrast showed chronic subocclusion of left internal JV with stenosis of the right internal Jugular vein.After being symptomatic for nearly 5 years, he had one severe in hospital headache episode with aphasia, right facial and arm paresis, and bradycardia (Cushing reflex).Intermittent intracranial hypertension from decompensation of decreased venous drainage of intracranial venous blood was diagnosed given the documented elevated severe elevation of intracranial pressure, the recurrent and reliable response to LPs, and bilateral jugular pathology.Urgent right JV stenting improved venous flow into the JV and reduced frequency and severity of headaches.He is presently on oral Aspirin and has returned to his regular sporting activities and founded a family.Conclusion: We postulate that physiological elevation of central venous pressure during exercise induced a vicious cycle of insufficient CSF drainage and intracranial hypertension, which decreased the size and absorption capacity of cerebral venous sinuses. . https://n.neurology.org/content/84/14_Supplement/P1.305

*"Spontaneous intracranial hypotension, of which brain slump is an extreme expression, is caused by a cerebrospinal fluid lea

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Can anyone explain why/suggest a resource that explains it well? You would think they both would have an absent descent because no blood can flow from RA to the RV.

Thanks.

I was under the impression that Diastolic BP was due to Systemic vascular resistance; and Systolic blood pressure was due to Stroke Volume.

Thus Arteriolar constriction should lead to Increase Diastolic BP, and Venous constriction should lead to increased Preload which should in turn lead to Increased SV and Systolic BP.

The Y descent on the jugular venous waveform represents atrial emptying, but why is it increased during restrictive cardiomyopathy or constrictive pericarditis? Wouldn't atrial emptying be inhibited given the stiffness of the ventricle leading to less of a visible descent?

I'm just confused because Y descent is blunted in cardiac tamponade, which makes sense given that the the ventricle is less capable of expanding and atrial emtpying is inhibited. Thank you guys so much!

Hello,

Thank you for reading this. I was studying a topic on CVPhysiology.com, and came across this excerpt on factors influencing central venous pressure:

https://preview.redd.it/1fohfthx0zs71.png?width=1650&format=png&auto=webp&s=f8233d4cf4be6c486ab5a9c4b5c2bfbce4213c76

• I don't understand the highlighted text. Would greatly appreciate any help with that.
• Also, as a hypothetical, let's say that α-1 receptors only caused vasoconstriction of arterioles—not both arterioles and veins. now, would α-1 activation cause decreased central venous pressure?

Thank you.

Isn't Venous Return supposed to decrease if Right Atrial Pressure increases?

Does anyone have a good understanding of venous sinus pressure and how it relates to IIH. I guess what I’m getting at is I have IIH and had an angiogram which showed my transverse sinus and jugular vein on right side is narrowed + my venous pressure was over 40 (supposed to be around 15?).

My neuroradiologist basically said the vp is likely from anxiety, try taking propranolol to lower it and maybe in a few years the veins will plump back up and pressure will go down. Wow, Ok.

The vibe I got from this doc was not good so I started searching online and found Dr. Patsalides and he has helped educate me more than anyone thus far. But, he doesn’t talk about vp and the role, if any, that plays.

I did learn that it’s common for one side of the sinus to not match the other and that’s perfectly normal. Does that mean since one side of my venous sinus is narrowed that it shouldn’t be causing problems? Eeesh, I get a little confused by all of it - hope my post makes sense to someone lol

Am having a brain fart rn....

So from what I know and the videos I have watched in inspiration thoracic cage expands and the diaphragm moves down creating abdominal pressure which opens Venous valves and helps venous return to the center of the body and as expiration occurs diaphragm relax back to place making that central venous pressure increase venous return to the heart and in result increase cardiac output . This is why Deep breathing exercises are so helpful for circulation. Now ... I came across this question in Uworld where A patient was in POSITIVE pressure ventilation intubation and the answer was that an adverse effect of this is Hypotension . The explanation says all the oposite from other resources where i learned venous return . It says that positive pressure applied to the lungs increase intrathoracic pressure during inspiration (Ofcourse) decreasing venous return and cardiac output and causing hypotension.

Am so confused .... Can someone explain this

Hi all, I am a medical student preparing for my exam in heart/lung physiology.

In one of the older exam papers I am using in preparation for my upcoming exam, it states that a pulmonary embolism will result in the CVP going up (which will lower the amount of venous blood flowing into the right atrium). It does not explain why this happens, and I am having trouble finding information on the internet (english is not my main language, and I could not find anything in my own language).

If anyone could explain to me why this happens it would be really awesome. Thank you in advance.

Had cat scan of head showed part of my jugular vein slipped into my right middle ear. Have visual issues and vs with all of its symptoms. I’ll keep you guys updated on more findings

Slide fom Boards & Beyond

And on the upper left here is an image showing it visually.

It doesn't make sense to me for tricuspid regurg to cause such a huge spike during atrial filling because it doesn't seem like there would be enough volume (since it's after systole).

I can see how in aortic regurgitation a compartment like the aorta could cause significant pressure changes in the LV since the aorta is always full, but I just don't get how tricuspid regurg can cause such a huge spike.

I mean, I get that not all the blood is ejected during systole (normal EF is >55%), but still.

Heart rate doesn't always increase that much. Sometimes its lower. There is always excessive pooling in my legs though which get very painful very quickly and take on a blotchy red hue or sometimes even blue blotchiness.

I generally feel pretty weak most of the time. When I don't and I do something like go to the grocery store I pay for it in pain and fatigue for the rest of the day, sometimes the next day too. I need help with a lot of things. Getting things or carrying my baby because I feel weak or lightheaded. Needing a lot of naps from fatigue. I am so fatigued within minutes after eating. I don't have much of an appetite so I eat about once a day. This blood pressure thing is kind of new. Mine usually runs in the 110s/70s range. Sometimes just a little more but not ever enough to be considered hypertensive. I also have a history of having blood pressure on the low end as well. Made me feel like crap so doc told me to add more salt to my diet. I did and now it kind seems all over the place.

Everything gets worse close to my period and for a while during and after. I have a lot of pain and heaviness in my legs, pelvis and lower back. I'm thinking the blood pools there too. I get out of breath a lot that is not proportional at all to what I'm doing. Gotta go pee? Out of breath. I feel like everyday all day is dictated by the level of pain, lightheadedness and heaviness that I feel. So badly to the point that I don't make plans anymore for anything because I never know how I'm going to feel. Sometimes my legs feel so heavy that even laying on my side one leg on top of the other hurts like my bones feel so heavy that they're just going to cut right through my skin.

It's worth noting that I'm 6 months postpartum and this all began in my 7th month of pregnancy. The only thing that was really different then was that if I sat up of stood up there was such an intense and painful pressure at the groin and top of my legs that it felt like they'd pop right off if I didn't get in some other position to let the blood flow which position was my left side. Like sitting up or standing would completely block the blood from leaving my legs and they'd very quickly turn blueish and within seconds I would be in unbearable pain.

So I don't know what the hell is going on with my body but I have been dealing with this for a long time now with no answers and I'm feeling so hopeless. My OB was a joke and didn't help me with any of this. She only treated me like I was ex

Did the bnb video, but the stuff disapears from my brain every 7 days. Any good mnemonics/memory hooks ?

I understand that an increase in resistance will cause a increase in pressure. So an increase in arterial pressure will cause a greater pressure difference between arterial and venous. Wouldn’t that mean a increase in venous pressure?

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