Do different nerves have varying distributions of afferent and efferent fibers in their composition? For example, the vagus nerve has about 80% afferent and 20% efferent vibers. Is this similar to other nerves? If not, what is the reason for this disparity? I understand that the vagus nerve plays a large part in the parasympathetic nervous system, but is this distribution similar in other nerves? There doesn't seem to be much information here (at least from my quick look at it).
Anyone here with nerve damage symptoms and with a small fiber neuropathy diagnosis? How are you doing? Anyone started to have their symptoms decrease over time (without being on pain medication)? I am taking nerve pain meds and vitamins / supplements . I wonder if I will ever be able to wear off the meds in my life.
Can a damaged peripheral nerve with sympathetic nerve fibers cause pots/dysautonomia?
Let's say somebody has a trapped peripheral nerve that has sympathetic nerve fibers. And let's say that person develops central sensitization from that compressed/damaged peripheral nerve, meaning their pain goes from local to central. Could that person develop dysautonomia or pots from a compressed peripheral nerve that has sympathetic nerve fibers?
Could a compressed peripheral nerve cause a lack of blood going back to the heart and upper body?
Do surgeons spend hours connecting every tiny piece one by one or does the body have a way of repairing and attaching parts together itself, or is my idea of the human anatomy just way off?
Hello everyone!
I have an anatomy exam coming up sometime from September to October, depending on when I am prepared and would like to know if anyone has any tips on remembering afferent/efferent fibers.
Our exam requires us to know where each part of the brain receives afferent fibers from and where it sends its efferents. I am starting to get a hang of most of CNS portion however having to mindlessly memorize what each specific part sends and receives seems a bit tedious and was wondering how people here might have done it before.
I am using anki to help learn them, but would wonder if anyone has any other resources as I have the entirety of human anatomy to master as well :)
Thanks in advance.
I had some samples taken from my leg to evaluate for small fiber neuropathy. Epidermal nerve fiber density was normal but sweat gland nerve fiber density was 3, low end of normal is 38.
Has anyone else had this test done or been diagnosed with sfn? It supposedly occurs in people with eds more frequently. Thanks for your info, you guys are awesome!
Hi there, 17 y/o here. I recently learned that women feel more pain on average than men, and I was just wondering if HRT effects this. I feel like I'm already a bit of a wimp and honestly the only thing that I'll miss when I transition is the extra muscle lol now I actually have to get it myself (excuse the tangent)
>Human muscular strength stems from two factors: a mechanical one (the myosine-actine fibers that contract) and an electrochemical one (the innervation that controls said fibers). If I recall correctly, humans allocate motoneurons and motor axons in a different way from chimpanzees or gorillas, trading finer motor control for brute power. A chimp is two to five times stronger than a human per unit weight, but doesn't have the same level of fine motor control.
So if you were a human with enough nerve fibers to lift two to five times your own weight, how can you do that without sacrificing your motor control?
I have read differing accounts regarding nerve regeneration. Some accounts indicate that tingling and itching can be signs of nerve regeneration while other accounts don’t mention anything about what the process feels like. I was just wondering if anyone had any thoughts or experiences.
25 male, it’s not visible or anything but recently my leg was cramping up high so I was massaging the area, I noticed there’s what feels like a little pea lump along a sting of what I assume is nerve. Doesn’t move or anything and feels hard and taught like muscle or nerve. It’s fairly small and more towards the anus end. Part of me wants to worry but the other part thinks this is just normal anatomy and I’m feeling a nerve or something. (Pushing near it causes a strange nerve sensation) is this likely nothing to worry about?
I am reviewing my notes on kindey physiology and the RAAS is confusing me. I have had one lecturer teach me that Angiotensin 2 vasoconstrictor effect is greater on the efferent than the afferent arteriole. This is to maintain the hydrostatic pressure in the glomerulus so renal function and GFR is maintained in situations like hypovolemia or hypotension. where renal perfusion is low. This explains why you should not give ACE inhibitors to patients with kindey failure since it will decrease their GFR even more. However another lecturer said that during a big sympathetic actication, example a significant bleedning, Angiotensin 2 acts more on the afferent arterioli in order to decrease filtration so more blood volume is retained during a time of bleedning. Both intuitively make sense but they are not compatible with each other. Only thing I can think of is that Angiotensin 2 renal effect varies depending on its concentration.
found this mnemonic for nerve fiber types if anybody needs it ,good luck
https://www.youtube.com/watch?v=eQgD1IBNmEE
https://preview.redd.it/f3ds9kk2lm171.jpg?width=640&format=pjpg&auto=webp&s=175f2bfbcdd49ef105e799b33cf09cd3149fac2d
Let's say somebody has a trapped peripheral nerve that has sympathetic nerve fibers. And let's say that person develops central sensitization from that compressed/damaged peripheral nerve, meaning their pain goes from local to central. Could that person develop dysautonomia or pots from a compressed peripheral nerve that has sympathetic nerve fibers?
Could a compressed peripheral nerve cause a lack of blood going back to the heart and upper body?
