Oxygen radical absorbance capacity Puns

Hi all,

I was doing some reading in the library surrounding the LH2/LH1 complexes of purple bacteria and LHCII complexes of higher plants. I started to note a lot of the chlorophylls in these complexes are rather close to each other to allow for efficient energy transfer of excited singlet states between complexes. However, I was also reading about adaptations for photoprotection such as the NPQ mechanism in LHCII and the role of carotenoids in this.

Does anyone care to elaborate on these interesting phenomena, references are appreciated.

Here are some papers I have read on the structure of these complexes:

https://www.nature.com/articles/374517a0

https://www.youtube.com/watch?v=6hKq5A__yrY

https://pubs.acs.org/doi/abs/10.1021/bi051097a

https://bmcplantbiol.biomedcentral.com/articles/10.1186/1471-2229-6-32

sciencedirect.com/science/article/pii/S0006291X07002537

Thanks in advance,

Cy

I tried both cardarine and meldonium and for oxygen Capacity and half life the winner is meldonium and less side effects COMPARED to cardarine

I found meldonium to open the lungs more every breath so this is great if your during hoghest intensity or low oxygen environments and half life is 2 days so its longer than cardarine

But cardarine is a better muscle builder and when during endurance with weights cardarine is the winner but i beloeve meldonium wins if you want to be lightweight as possible as greater oxygen output + its cbeaper to buy.

What are your guys opinion?

Hi guys I bought the game 3 days ago for switch and have really been enjoying it so far !

Unfortunately I had a problem while saving and lost a whole lot of progress.

I managed to get everything I lost back except for the ultra high capacity oxygen tank. I don't have the blue print anymore but the "chest" where I found it is already opened (and empty) ... Do you know if there is an alternative location to get that blueprint back ?

https://twitter.com/himantabiswa/status/1386623068542439426?s=21

Developing Type-I photosensitizers are considered as an efficient approach to overcome the deficiency of traditional photodynamic therapy (PDT) for hypoxic tumors. However, it remains a challenge to design photosensitizers for generating reactive oxygen species by the Type-I process. Herein, we report a series of α,β-linked BODIPY dimers and a trimer that exclusively generate superoxide radical (O2−•) by the Type-I process upon light irradiation. The triplet formation originates from an effective excited-state relaxation from the initially populated singlet (S1) to triplet (T1) states via an intermediate triplet (T2) state. The low reduction potential and ultralong lifetime of the T1 state facilitate the efficient generation of O2−• by inter-molecular charge transfer to molecular oxygen. The T1-S0 energy gap is smaller than that of O2 thereby precluding the generation of singlet oxygen by the Type-II process. The trimer exhibits superior PDT performance under the hypoxic environment.

https://ift.tt/3hOq2Er

This is a pic of that notification.

I came across this on twitter. Some news website have reported this as well.

https://www.news18.com/news/india/covid-19-govt-to-allow-industrial-manufacturers-to-produce-oxygen-for-medical-use-2568781.html

https://wap.business-standard.com/article/pti-stories/covid-19-govt-to-allow-industrial-manufacturers-to-produce-oxygen-for-medical-use-120040701723_1.html

Many in the right-wing is using this to blame the states for oxygen shortage. Hence, my question in the title.

Amplifying free radical production by chemical dynamic catalysis to cause oxidative damage to cancer cells has received extensive interest for cancer-specific therapy. The major challenge is inevitable negative modulation on the tumor microenvironment (TME) by these species, hindering durable effectiveness. Here we show for the first time an oxygen vacancy - rich Bi-based regulator that allows environment-adaptive free radical catalysis. Specifically, the regulator catalyzes production of highly toxic O 2 •- and • OH in cancer cells via logic enzymatic reactions, yet scavenges accumulation of free radicals and immunosuppressive mediators in TME-associated noncancerous cells. Atomic-level mechanistic studies reveal that such dual-modal regulating behavior is dominated by oxygen vacancies that well fit for free radical catalytic kinetics, along with distinguished cellular fates of this regulator. With this smart regulator, a “two birds with one shot” cancer dynamic therapy can be expected.

https://ift.tt/3wFzHTN