Hoping my 3+ years of random trial and error can help someone else who maybe is like me: 47, Chinese, perimenopausal, combination skin, high-stress. PD for me is under my nose and along the sides in the crevices, outside the corners of my mouth, just under my lower lip. Redness, dryness, sometimes tiny red pustules that hurt.
I use Jason toothpaste, no SLS anywhere.
I finally am seeing a change by going with simple products. I have tried so many things. Most recently, I ordered some expensive miracle regimen from Australia (Amperna) and that didn’t work either. Avene ciclifate makes it worse for me and causes pustules to form, and sometimes whiteheads.
I have a regimen that has really transformed the texture, tone, clarity of my non-PD areas and I am loathe to give that up bc it cleared up my monthly cyclic acne and my face no longer feels reactive and puffy and itchy everywhere.
That regimen mostly avoids my PD areas. I was washing with gentle cleansers which seemed fine: Avene and Dermalogica gel but just switched to Natural Elephant Dead Sea sulfur soap. In the morning and evening, I use Tula probiotic toner all over, and their clearing spot treatment all over except PD areas. In the morning, I use Skinceuticals C&E Ferulic, and in the evening, Marie Veronique Intensive Repair Serum, along with Sunday Riley Luna oil, avoiding the PD areas. I have used various mild creams to nothing on the PD areas and nothing works.
I just got a tube of 20% zinc oxide from Amazon and so now am using sulfur soap, Tula toner, and the zinc oxide cream on my PD. The pustules are gone! My bumps are drying and healing. The flaking is mostly gone. I have a little redness left and the Ordinary’s alpha arbutin helps with that.
Fingers crossed this continues!
The reaction of hexachlorophosphazene, P3N3Cl6, with SO3 leads to the new sulfur nitride‐oxide S6N2O15. The compound displays an extraordinarily low nitrogen content and exhibits a bicyclic cage structure according to the formulation N{S(O)2O(O)2S}3N, with both nitrogen atoms in trigonal planar coordination of sulfur atoms. Interestingly, the new nitride‐oxide can be also seen as the anhydride of nitrido‐tris‐sulfuric acid, N(SO3H)3.
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Any dentists or whoever on here know the answer?
I have struggled with debilitating symptoms related to this gene mutation (SDHB). I notice my symptoms nearly disappear when I drink beet juice. I researched more about beets and I see they cause nitric oxide release. Then when researching the Krebs cycle, I see something about iron-sulfur proteins like SDH being destroyed by nitric oxide. The whole Krebs cycle is of course complicated for a non-chemist and I would love for someone to explain to me this interaction of nitric oxide and iron-sulfur proteins.
These are the 3 very different explanations I get after consulting experts in geoengineering, public health, and special ops.
Note each cites a totally different chem being sprayed.
Compartmentalized Disinfo? The great divide.
What are they NOT telling us, though? =/
We all know that nitric oxide (NO) is spontaneously oxidized by oxygen (O2), but what if sulfur dioxide (SO2) was also present?
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Does anyone know what the intermediate(s) are when nitric oxide reacts with diatomic oxygen (at room temperature)? What are the reaction mechanisms?
As anyone who knows about kinetic theory knows, it is exceedingly unlikely that any three molecules will simultaneously react with eachother. Nearly all reactions involve two molecules first reacting with eachother to form an intermediate.
In the case of one molecule of NO and one molecule of O2, any intermediate molecule or molecules that forms would be highly reactive.
There seems to be only two possible pathways:
Two molecules of nitric oxide can transiently dimerize into N2O2, which could then be oxidized with with a molecule of O2, presumably forming an intermediate square ring. The ring would then immediately break apart into two molecules of NO2.
One molecule of NO could be directly oxidized by a O2 to form a nitrate radical NO3•, which would then eventually react with another molecule of NO.
somewhere in the literature (if remember correctly) it stated that the reaction proceeds through both pathways, but that the first predominates, which might explain the relatively slow reaction rate (the reaction between the two gases is extremely rapid, but not quite instantaneous). but if even a small part of the reaction proceeds through the second path, could not the nitrate radical be used as a powerful oxidizer for some third regent?
For example, if sulfur dioxide was also introduced into the reaction at the same time?
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This may be somewhat speculative, but I was thinking the reactions might be:
O*2* + SO*2* + (2)NO --> (NO)2SO4
(4)O*2* + (4)SO*2* + (6)NO --> (2) (NO)2S2O7 + (2)NO*2*
(2)O*2* + (2)SO*2* + (2)NO --> (NO)S*2O7(NO2*)
The oxygen and sulfur dioxide should probably be pre-mixed, with the sulfur dioxide in large excess, then a much smaller portion of nitric oxide should be very slowly and gradually added. In any case, the oxidation of SO2, if it occurs to any extent, is likely to be very inefficient. Care should be taken to avoid the presence of any moisture, otherwise any oxidation products that might form would hydrolyze to sulfuric acid.
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(the chemical name for (NO)S2O7(NO2) would be nitrosyl nitryl pyrosulfate, and would likely exist as a crysta
I have struggled with debilitating symptoms related to this gene mutation (SDHB). I notice my symptoms nearly disappear when I drink beet juice. I researched more about beets and I see they cause nitric oxide release. Then when researching the Krebs cycle, I see something about iron-sulfur proteins like SDH being destroyed by nitric oxide. The whole Krebs cycle is of course complicated for a non-biochemist and I would love for someone to explain to me this interaction of nitric oxide and iron-sulfur proteins.
