A list of puns related to "St Elevation"
55-year old male, 6 feet, 183 pounds, white. Currently take an anticoagulant (Eliquis) to prevent venous thromboembolism. (I've had deep vein thromboses on two separate occasions over the last four years.) Do not drink or smoke tobacco. Smoke cannabis a few times per week.
I recently had an ECG as a part of a routine check up. The interpretation is "Sinus rhythm -- Inferior ST elevation -- Early repolarization." The remainder of the report reads as follows:
T wave axis: 38 deg
QRS axis: Β 4 deg
P wave axis: 59 deg
QRS duration: 102 ms
QT interval: 394 ms
PR interval: 178 ms
Heart Rate: 66 BPM
I'd greatly appreciate it if someone could let me know whether the finding of an "Inferior ST Elevation -- Early Repolarization" is a normal variant for someone my age (i.e., 55 years old), or whether it indicates a potential problem and should be followed up on. Thanks!
Good evening,
I've been researching this for hours now and I just can't understand it. My textbook states that ST-elevations or -depressions can point towards different things, depending on whether they are concordant or discordant with the vector of ventricular depolarisation.
Apparently a concordant ST-elevation or -depression would point towards subepicardial damage (e.g. acute infarction or pericarditis), while a discordant one would point towards subendothelial damage (e.g. coronary artery disease or hypertrophy).
However what my textbook doesn't explain and what I still can't wrap my head around: How does this occur?
I've read about discordant ST-depression in left ventricular hypertrophy occuring due to prolonged repolarisation, but this confuses me even more. Prolonged repolarisation shouldn't affect the isoelectric ST-segment (since in my understanding, full depolarisation can still occur), so how is this even connected?
I'm hesitant to ask my professor about this since the last time we talked about ECGs was about a year ago, and back then this question didn't occur to me, so asking it now would be pretty awkward. I'm hoping maybe you could help me understand this.
Thanks a lot <3
Anyone know or wanna speculate the elevation change on 9th st. between Amazement Square and the courthouse? Google maps is telling me it's about 190ft, the stairs at monument terrace have containing roughly 70 ft of that of that 190 total.
I was thinking it was more that 190ft.
I remember reading it some where in the uworld but now i cant find it.
Can anyone explain why the EKG records a depression when there is a subendothelial infarct and a ST elevation when there is a transmural infarct?
I've been doing some research into the ECG changes in acute pericarditis and am struggling to find a decent explanation for the widespread ST Elevation in the disorders initial stages.
The explanation in this video https://www.youtube.com/watch?v=VqY-lK9A7SY is good and makes sense from a pathological point but I can't as yet find anything to back this information up.
Does anyone have any resources that go into the pathology of the ECG changes in pericarditis?
Anyone can explain the following flashcard? Thanks!
https://preview.redd.it/yq4wlwrc2pf61.png?width=2292&format=png&auto=webp&s=8f781c3f58fa482ae1ba6fbaa7b46a20c13ea18b
Hi y'all,
Full disclaimer: I'm super new to EKGs.
This might be a dumb question, but we recently had a pt whose 4 lead showed obvious ST elevation in lead II. When I put the full 12 lead, there was no ST elevation in any of the leads.
I asked my partner about it and he said it has to do with the filters added to 12 lead readings on the monitor, but I couldn't find a lot of specifics when I tried to look up why this would happen.
Could someone explain why there was such a huge difference between the readings? Thanks!
(In case it's of any importance, pt was a 49YOM and CC was severe indigestion)
Iβm a paramedic student trying to research the exact cause of ST segment changes in MI. I comprehend how the changes relate to location of the ischemia/infarction, but I cannot find a credible resource explaining the cellular mechanism behind the changes in amplitude ST segmentβs amplitude. I greatly appreciate any information or resources available!
https://preview.redd.it/izzdmqi42lk51.png?width=1684&format=png&auto=webp&s=6c20cfaf92c19bb7ee5a84c822c6aa258bf76a7e
https://preview.redd.it/4t7y00d62lk51.png?width=1680&format=png&auto=webp&s=c79d1e0000d02da7b8c400c334dd1ce1be2fdf83
Please note that this site uses cookies to personalise content and adverts, to provide social media features, and to analyse web traffic. Click here for more information.